by Rhoda Wilson, Expose News:
Dr. Robert Malone published an essay last month detailing why most of what we’ve been told about annual influenza vaccination is propaganda.
Revelations in his essay include increasing annual influenza vaccination of the elderly is associated with increased influenza-related death in the elderly, the flu vaccine can make you more susceptible to influenza-like illness, deploying leaky vaccines (into either humans or poultry flocks) will accelerate the evolution of vaccine-resistant influenza viruses and most of the US government (“USG”) annual influenza vaccination policy is influenced by a desire to support and maintain influenza manufacturing capacity.
He also discussed the 1918 “Spanish flu” pandemic. Most Spanish flu deaths, he said, could have been prevented if antibacterial medications, or antibiotics, had been made available.
TRUTH LIVES on at https://sgtreport.tv/
The following is extracted from the article ‘Deconstructing HHS: Influenza Vaccine Propaganda’ written by Dr. Robert Malone and published on 10 February 2025.
Let’s examine the influenza vaccine heresies one by one.
Table of Contents
Increasing Annual Influenza Vaccination of the Elderly is Associated With Increased Influenza-Related Death in the Elderly
The main issue with influenza is “influenza-like” disease and death (morbidity and mortality) in the elderly and in those with significant pre-existent conditions. In other words, upper-respiratory viral pneumonia on top of certain other conditions can tip the sick and elderly over the edge. This is similar to the issue of disease and death from SARS-CoV-2 (covid) primarily happening to people that had other health problems – one of those other health problems being old age in general. People mostly died WITH covid, not necessarily FROM covid – same with influenza. And by the way, “influenza-like illness” is a grab bag.
For purposes of public health data analysis, in most cases, upper respiratory viral disease is typically assumed to be due to the influenza virus. The inconvenient fact is that there are many viruses and other pathogens that cause “influenza-like” illness, disease and death. Influenza viruses (types A and B), respiratory syncytial virus (“RSV”), parainfluenza viruses, rhinoviruses, coronaviruses, adenoviruses, metapneumovirus, group A streptococcus, mycoplasma, chlamydia and Bordetella pertussis. The most common cause of “walking pneumonia” is mycoplasma – which is not really a virus! From this brief explanation, you can see that the cited “deaths from influenza” data are usually inflated, just like “deaths from covid” were.
There is a clinical saying, that “pneumonia is the old man’s friend.” This means that when you are old, feeble and suffering from various chronic disease conditions, a quick death associated with pneumonia (often with sepsis complications) can relieve you from pain and suffering.
Of course, now we have government-supported MAiD (medical assistance in dying) in many countries which provides an even easier, less traumatic exit for those who are not concerned about the theological, ethical, practical or conflict-of-interest implications of state-sponsored medical suicide.
All market-approved influenza vaccines include or encode proteins (antigens) from both Influenza A and Influenza B. The specific strains of A and B included in a given year’s vaccine preparation vary from year to year based on recommendations from a WHO working group that looks at northern and southern hemisphere trends modelled based on preceding year strain monitoring data.
Now you have the background to better appreciate this bombshell “peer-reviewed” paper.
Abstract
Background: Observational studies report that influenza vaccination reduces winter mortality risk from any cause by 50% among the elderly. Influenza vaccination coverage among elderly persons (> or =65 years) in the United States increased from between 15% and 20% before 1980 to 65% in 2001. Unexpectedly, estimates of influenza-related mortality in this age group also increased during this period. We tried to reconcile these conflicting findings by adjusting excess mortality estimates for ageing and increased circulation of influenza A(H3N2) viruses.
Methods: We used a cyclical regression model to generate seasonal estimates of national influenza-related mortality (excess mortality) among the elderly in both pneumonia and influenza and all-cause deaths for the 33 seasons from 1968 to 2001. We stratified the data by 5-year age group and separated seasons dominated by A(H3N2) viruses from other seasons.
Results: For people aged 65 to 74 years, excess mortality rates in A(H3N2)-dominated seasons fell between 1968 and the early 1980s but remained approximately constant thereafter. For persons 85 years or older, the mortality rate remained flat throughout. Excess mortality in A(H1N1) and B seasons did not change. All-cause excess mortality for persons 65 years or older never exceeded 10% of all winter deaths.
Conclusions: We attribute the decline in influenza-related mortality among people aged 65 to 74 years in the decade after the 1968 pandemic to the acquisition of immunity to the emerging A(H3N2) virus. We could not correlate increasing vaccination coverage after 1980 with declining mortality rates in any age group. Because fewer than 10% of all winter deaths were attributable to influenza in any season, we conclude that observational studies substantially overestimate vaccination benefit.
Simonsen L, Reichert TA, Viboud C, Blackwelder WC, Taylor RJ, Miller MA. Impact of influenza vaccination on seasonal mortality in the US elderly population. Arch Intern Med. 2005 Feb 14;165(3):265-72. doi: 10.1001/archinte.165.3.265. PMID: 15710788.
In other words, according to this study, natural immunity acquired by influenza infection works to prevent future “influenza-related” deaths in the elderly. Vaccination to a specific influenza A virus strain (H1N1) does not improve natural immunity to that specific influenza A virus strain and, on average, increased influenza vaccine uptake increases all-cause “influenza-related” death (mortality) in the key age group of the elderly, where most of the “influenza-related” deaths occur.
This is not what we are told, and it calls into question whether we are wasting a lot of money and effort (and propaganda) to dose the entire population with injectable products that DO have risks. Whatever those risks are, how serious, how frequent, in what age and risk factor groups we do not really know because this is (basically) a forbidden topic of inquiry.
Maybe MAHA (Make America Healthy Again) should think this through again?
Annual Influenza Vaccination Can Make You More Susceptible to Influenza-Like Illness
The issue here was originally described as “original antigenic sin” but now the more politically correct term is “immune imprinting.”
The term “original antigenic sin” (“OAS”) was first used in the 1960s to describe how one’s first exposure to influenza virus shapes the outcome of subsequent exposures to antigenically related strains.
The “cliff notes” version here is that if you get “boosted” every year with a suboptimal influenza “vaccine,” it sets your immune system up to be focused on last year’s virus rather than to being better able to respond to tomorrow’s strain. This is a form of immune system bias. This can make it so that you are less able to fight off newly evolved strains.
Maybe MAHA should think this through again?
This leads directly to the next point.
Deploying Leaky Influenza Vaccines (into Either Humans or Poultry Flocks) Will Accelerate the Evolution of Vaccine-Resistant Influenza Viruses
“Leaky vaccine” is industry jargon for “partially effective products” intended to prevent infection, replication, spread and disease caused by what you are “vaccinating” against. As can be inferred from the initial paper cited above, the current gold standard for “effectiveness” in influenza “vaccines” is natural infection. And natural infection is not completely effective. Otherwise, we would all get one influenza A infection and one influenza B infection when we are children and that would confer lifelong protection against all influenza viruses.
Influenza virus continues to circulate in humans (and birds, and other animals) because it is partially able to evade immune responses generated by prior infections in these animals. And it continually evolves (“drifts and shifts”) to better evade those responses.
The less effective a “vaccine” product is at preventing infection and replication of an infectious pathogen, the more likely widespread dosing with that product will select for pathogens that are more ‘vaccine resistant.” This, plus the fact that “bird flu” is endemic in wild birds, is why we CANNOT vaccinate our way out of the risk posed by “bird flu” to either poultry flocks or other animals. If we vaccinate commercial flocks (say ducks or chickens) with a partially effective vaccine, what we will get is “bird flu” that has evolved to become more resistant to that vaccine. Furthermore, “vaccination” that partially suppresses disease without preventing replication and spread of the virus will actually increase the risk of crossover into the humans that handle those birds because they will have a harder time recognising a sick flock and so will be less likely to take precautions to not get infected themselves.
You cannot “vaccinate” your way out of an outbreak of influenza (or a coronavirus, for that matter) with an imperfect “vaccine,” and if you try you will only make matters worse. This is a fundamental truth. All influenza vaccines to date are imperfect because we have not learned how to make a “vaccine” that is sterilising and works better than natural infection.
Maybe MAHA should think this through again?
The Real Problem With Influenza Virus-Associated Death in the Elderly is Due to the Ageing of Their Immune Systems (Immunosenescence)
Yes, as we age, for some reason, our immune systems age with us. Immunology scientists like to create their own words and language for everything in their field (I like to call it “immunobabble”); usually coining words and phrases that include some part of the term ‘immunology.” So, it is no surprise that when they encountered the reality that immune systems become less effective as they age, they coined a term merging “immuno”- and a fancy term for ageing – “senescence.”
Infection susceptibility, poor vaccination efficacy, age-related disease onset and neoplasms are linked to innate and adaptive immune dysfunction that accompanies ageing (known as immunosenescence).
Liu, Z., Liang, Q., Ren, Y. et al. Immunosenescence: molecular mechanisms and diseases. Sig Transduct Target Ther 8, 200 (2023). https://doi.org/10.1038/s41392-023-01451-2
If we are going to shift the NIH research enterprise to focus on promoting health rather than preventing and treating specific diseases, including influenza-like illness, maybe we should focus the hundreds of millions of dollars being spent on developing an mRNA vaccine for “bird flu” to understanding the causes of immunosenescence. Because it looks like many of those causes are also involved in a wide range of other diseases – including cancer.
