by Richard Gale and Dr. Gary Null, Global Research:
Efforts to eradicate polio, once considered a public health triumph, are now threatened by a complex interplay of scientific, logistical, and geopolitical challenges. The emergence of vaccine-derived polioviruses (VDPVs) and a rise in vaccine-related complications have introduced unforeseen obstacles. Despite the decades that our federal health officials believed mass vaccination campaigns were making progress, polio cases remain a persistent threat driven by socio-political factors, vaccine-associated risks, and new strains that may have very likely developed from over-vaccination. Research from leading institutions such as Stanford University, the CDC, and global health organizations like the World Health Organization (WHO) has increasingly confirmed independent researchers’ long-held concerns that the polio vaccines’ success story has been overblown.
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The WHO in earnest launched its global polio eradication initiative in 1988. In 2002, Europe declared itself to be polio-free. In 2015, the WHO declared wild poliovirus type 2 was eradicated globally. However, recent detections of vaccine-derived poliovirus in Europe and the US, outbreaks in the Philippines, and resurgent cases across Africa and Asia have proven that vaccine-derived herd immunity has been nothing more than a myth.
For example, earlier this year researchers from the universities of Essex and Cranfield in the UK discovered poliovirus in wastewater samples from Germany, Spain, and Poland. Although no clinical cases were reported, the detection is worrisome. It signals the ongoing circulation of the virus. These findings highlight how the live attenuated virus in the oral polio vaccine (OPV) may have mutated and regained virulence. The British researchers noted that the strains identified were linked to vaccine-derived poliovirus. These detections parallel earlier outbreaks in Tajikistan and Ukraine in 2021 and Israel in 2022, where vaccine-derived strains caused paralytic disease. In the backdrop of the genocide against Palestinians, polio cases are reaching epidemic proportions in Gaza. In the UK, poliovirus was similarly detected in wastewater in 2022.
Vaccine-derived polioviruses arise from the oral polio vaccine, which contains a weakened live viral strain. Under certain conditions, it can mutate and regain its virulence. It is particularly dangerous in regions with poor sanitation where it spreads through fecal contamination. These mutated strains then behave like wild poliovirus and cause paralytic disease. This problem with the oral polio vaccine was first documented in 2000 and has gained prominence with the increase in polio vaccination campaigns. For instance, during the first nine months of 2014, Nigeria recorded more polio cases caused by vaccine-derived polioviruses than wild poliovirus. In 2019, the Philippines declared a polio outbreak nearly two decades after being certified polio-free by the WHO. A case was confirmed in a three-year-old girl in Lanao del Sur province followed by poliovirus being detected in sewage samples from Manila and waterways in Davao. Genetic sequencing has also linked these outbreaks to cross-border transmission fueled by population movements. In 2020, Sudanese children were being paralyzed by vaccine-derived poliovirus type 2 that was traced to an outbreak originating in Chad. Although the live polio vaccine has been discontinued in developed nations, due to its risks in causing polio-like paralysis, it is still administered in poorer undeveloped countries. As long as the drug companies continue to make and distribute oral polio vaccines, solely for its cost saving benefits and ease to manufacture, vaccine-induced paralysis will rise. These outbreaks attributed to VDP type 2 highlight the risks associated with oral polio vaccine in areas with inadequate sanitation and water utility systems. Dr. Walter Orenstein, a professor of medicine at Emory University and former director of the US National Immunization Program has described vaccine-derived polioviruses as “the biggest surprise” in global vaccination efforts.
Today, US health authorities proudly claim the nation is polio-free. Medical authorities and advocates of mass vaccination raise the polio vaccine as an example of a vaccine that eradicated a virus and therefore proof of the “herd immune theory.” The simplistic belief that regional eradications of polio are an exemplary model of vaccine-induced herd immunity applicable for all other vaccines is both naïve and dangerous. It is a propagandist narrative that won’t disappear. It was parroted during the Covid-19 pandemic. For example, a University of Otago study claimed that mass Covid-19 vaccinations would achieve even better results than the polio vaccine. The entire one-size-fits-all study was fundamentally flawed. The New Zealand scientists’ claim that COVID-19 eradication might be feasible, and even more achievable than polio eradication, is fundamentally flawed. By drawing comparisons between COVID-19 and diseases like polio, the study overlooked critical distinctions in viral behavior, vaccine efficacy, and risk-benefit ratios due to adverse effects. It also failed to account for the fact that neither the Pfizer nor Moderna mRNA vaccines are capable of preventing infection, transmission and viral shedding.
Image: Copyright: AP
In light of the growing life-threatening risks associated with Covid-19 vaccines, it has become abundantly clear that fast tracking unsafe and poorly researched vaccines is a disastrous public health policy. This was also the case for one of the first polio vaccines in 1955. In fact, the polio vaccine received FDA approval and licensure only after two hours of regulatory review – the fastest approved drug in the FDA’s history. Known as the Cutter Incident, named after the vaccine’s manufacturer Cutter Laboratories, within days after its first mass administration, 40,000 children were left with polio, 200 with severe paralysis and ten deaths. Shortly thereafter the vaccine was quickly withdrawn from circulation and abandoned.[1]
Modern medical wisdom believes that the enterovirus associated with poliomyelitis is a highly infectious disease. The virus enters the body’s system through the gastrointestinal tract, often because of fecal contamination in water resources. According to the CDC, the majority of people who contract this enterovirus will not experience any symptoms. Approximately 25% will show temporary flu-like symptoms that disappear after several days. It is only after the virus enters the bloodstream and infects alpha-motor neurons located in the spinal cord’s anterior gray matter that there is the risk of paralytic poliomyelitis.
A question that has plagued historians of medicine is whether or not the scourge of paralysis starting in 1916, well before the introduction of the first polio vaccine, was actually caused by the virus. Before 1916, when there was an enormous leap in paralysis cases in a single year—from zero to 3 cases per 10,000—why were there no recorded cases in the medical literature before that time? Moreover, after a couple years, the cases declined back to near zero. For the next 35 years, there would continue to be spikes and declines until 1948 when polio cases started to skyrocket.
The enterovirus theory has never provided a satisfactory answer to account for poliomyelitis’ sporadic trends. In a recent article about the Rockefeller Institute’s role in fraudulent research to identify the pathogen behind the rise in poliomyelitis, William Engdahl presents a 1909 paper published by Rockefeller employees Simon Flexner and Paul Lewis whereby they assert the poliomyelitis virus had been identified after injecting diseased human tissue into monkeys, who then developed symptoms. However, Flexner and Lewis admitted they found no bacteria or conclusive evidence of a virus. They acknowledged, “We failed utterly to discover bacteria… and we had failed to obtain any such bacteria from the human material studied by us;” yet, they concluded the cause “must” be a virus, despite lacking any scientific proof. Their experiment involved using impure, contaminated materials, including spinal cord, brain, and even fecal matter, injected into monkeys. This speculative leap was accepted by the medical world without scientific proof, and the existence of an enterovirus as a paralytic agent remained unverified until 1955.
In his Vaccines, Autoimmunity and the Changing Nature of Childhood Illness, Dr. Thomas Cowan identifies some odd coincidences during the periods when paralysis cases spiked. For example, the first cases of paralytic polio clustered around Coney Island in New York, and later started to appear in the larger cities of the Northeastern states, such as Boston, Philadelphia and Baltimore. Curiously, the two major spikes in poliomyelitis cases — 1916 to 1918, and 1948-1955 — correlate with the widespread use of two dangerously neurotoxic chemicals: arsenite of soda and dichloro-diphenyl-trichloroethane, commonly known as DDT.
At the end of the 19th century, the majority of sugar came from Hawaii’s sugarcane plantations. The industry was in a crisis due to weed proliferation. A plantation owner, Charles Eckhart, came up with the idea of spraying fields with a very potent form of arsenic known as arsenite of soda. Arsenic is also highly toxic to anterior horn cells, motor neurons that protect gray matter in the spinal cord. Over the years, sugar entering the US mainland was heavily contaminated with this neuro-toxin. The first recorded case of so-called polio was noted in Sweden where an arsenic-based insecticide was employed, and the famous Vermont polio outbreak several years later happened in a region where a lead arsenite spray was used to eradicate gypsy moths. Around this same period, the Swiss scientist Paul Hermann Muller first synthesized DDT in 1874 and started to be used as an insecticide in Europe to combat gypsy moths.
However, the largest rise in polio cases occurred when DDT was indiscriminately sprayed across large regions of the US. Older generations will remember television scenes of children literally being sprayed down with the chemical. Not all scientists were convinced that the epidemic of paralytic cases was being caused by a virus. Dr. John Polyani, who would later receive the Nobel Prize in chemistry, opposed the hypothesis because it failed to meet Koch’s postulates: 1) a pathogenic organism must be present in every case of disease, 2) that the pathogen must be isolated from a disease host, and 3) the disease must be reproduced when introduced into a healthy host. These postulates have never been completely validated for an enterovirus as being the causal agent of poliomyelitis.
