by 2nd Smartest Guy in the World, 2nd Smartest Guy in the World:
There has been a lot of deliberate disinformation of late about the miracle drug Ivermectin, which has now been handily debunked by actual experts.
First off, Dr. Tess Lawrie brilliantly responded to the imbecilic attacks in the following article:
TRUTH LIVES on at https://sgtreport.tv/
And then Dr. Lawrie did an astute followup on the continent that has been regularly administering Ivermectin with not only no losses in fertility, but, rather, increases in fertility, as well as innumerable lives saved thanks to this life-saving drug:
It is now painfully obvious that Dr. Mike Yeadon, former vice-president of Pfizer’s allergy and respiratory research, is a BigPharma shill that after decades of virus research suddenly comes to the conclusion that all of the virions that he was studying never existed, and as such there never were any viruses to begin with! How convenient. He also recently admitted that he did not know what vitamin K2 was. Etc. & etc.
In honor of shills like Dr. Yeadon, and the various know-nothing fear peddlers there is a FLASH WEEKEND SALE, but first here is a very important additional article taking down Dr. Yeadon and his inane claims:
June 2024 Interview (above video) with Dr. Mike Yeadon, former Vice President of Pfizer
In a June 2024 interview, Former Pfizer VP Dr. Mike Yeadon, a strong advocate for medical freedom and advocate for the lack of evidence of SARS-CoV-2’s existence, stated “Ivermectin is one of the most violent anti-fertility drugs i’ve ever encountered… unquestionably, at the doses you’re being suggested to take… in studies in rats, rabbits, and limited studies in humans, it reduces the ability to conceive and grow babies to term.”
LETS LOOK AT THE EVIDENCE, SHALL WE?
MICE:
2018 Sweify et al – The effect of ivermectin on secondary spermatocytes and serum total proteins
- The present work searched for the potential mutagenicity of IVM in mice. This was achieved by using cytogenetic and total protein content.
- Male mice treated with single injections of 200ug/kg b.w. IVM
- IVM is produced a considerable signs of chromosomal damage to germ cells.
- So, the cytogenetic studies revealed high clastogenicity of the drug.
- On the other hand, the differences in total protein concentration obtained between treated and control samples indicate genotoxic potential for IVM.
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RATS:
2008 El-Nahas – Effect of ivermectin on male fertility and its interaction with P-glycoprotein inhibitor (verapamil) in rats
- The results revealed that administration of ivermectin once weekly for 8 weeks induced slight fertility disturbances
- Cytogenetic study revealed partial effect of ivermectin on meiosis
- We concluded that ivermectin has slight effects on male fertility,
2018 Cordeiro et al – Ivermectin acute administration impaired the spermatogenesis and spermiogenesis of adult rats
- IVM acute exposure, mainly in the higher dose, reduced the testicular volume, the tubular diameter and the germinal epithelium height
- histological studies show that tubular sections containing several histological changes indicative of spermatogenesis interruption, such as disorganization of germinal epithelium, vacuolar degeneration of the germ cells and sloughing of cells into the tubular lumen
- no differences in testosterone levels
- It was concluded that acute IVM impaired the spermatogenesis and spermiogenesis of rats
- Our results suggest that some testicular effects are reversible and correlated with the plasmatic levels of IVM.
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RABBITS:
2001 Emam – Effects of ivermectin and moxidectin on fertility and some biochemical parameters in male rabbits.
- An experiment was conducted to evaluate the effect of injecting a therapeutic dose (0.2 mg/kg body weight) of ivermectin (Ivomec) on the sperm characteristic, serum testosterone level and some blood biochemical constituents of male rabbits
- Treatments had an adverse effect on fertility of rabbits at 30 and 60 days post-injection, and significantly decreased sperm cell concentration percentages of progressive motility and live sperm, and serum testosterone levels. In comparison, the percentage of total sperm abnormalities was significantly increased.
- The induced hazard effects by the tested drugs on these parameters were reversible as they returned to normal values after 90 days of the experiment.
2019 Moreira – Ivermectin does not interfere with seminal and hormonal parameters in male rabbits
- this study evaluated the temporal (1 up 60 days) effects of exposure to IVM (0.2 and 1.0 mg/kg, administered subcutaneously) on seminal and hormonal parameters of male rabbits
- Ivermectin did not impair spermatozoa motility and morphology.
- Ivermectin was not injure the plasma and acrosomal membranes and neither alter the mitochondrial potential of spermatozoa.
- Ivermectin did not interfere with the levels of serum testosterone.
- Ivermectin did not alter serum hematological and biochemical parameters.
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EWES
2021 Abdel-Rahman et al – Clinicopathological and Reproductive Studies on The Use of Ivermectin in Ewes
- This study aimed to investigate the effect of Ivermectin (IVM) on the reproductive hormones and hemato-biochemical parameters of twenty apparently healthy ewes weighing 30-40 kg and 2-3 years old.
- treated with the recommended therapeutic dose of IVM (0.2 mg/kg, S/C) one day after parturition (treated group)
- It has been concluded that IVM delayed estrous in ewes for three months via disturbing the female reproductive hormones and the hemato-biochemical parameters. Therefore, it is recommended that IVM not be injected at least three months before the reproductive season.
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IRAQI AWASSI RAM
2012 Naoman – Effect of Ivermectin on semen characteristics of Iraqi Awassi ram
- subcutaneously injected with ivermectin at dose 0.2 mg/ kg
- Result of this study shows significantly (P<0.05) decrease in semen volume, sperm concentration and serum testosterone levels of treatment group while percentage of mass and individual motility show no insignificantly differences after 3 days following ivermcetin injection and Sperm abnormalities increase significantly (P<0.05) in treatment group comparing with the control group.
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