by Eric Zuesse, The Duran:
On 27 February 2023, the youtube “Transparently Hiding…Again” “David Martin World” appeared, and I here give not only its link but the link to the first web-archived copy of it so that if the original version gets yanked, you’ll still be able to see and hear it, because it not only asserts but proves that covid-19 was engineered by the U.S. Government in North Carolina to be developed in Wuhan China, instead of in the United States, so that China would get the blame. Here is that youtube:
TRUTH LIVES on at https://sgtreport.tv/
https://www.youtube.com/watch?v=CMSz209wV8g
https://web.archive.org/web/20230228203633/https://www.youtube.com/watch?v=CMSz209wV8g
“Transparently Hiding…Again” “David Martin World”
27 February 2023
I carefully accessed online each one of its documentary sources, each of which was displayed in the video, and found that each statement that Mr. Martin was saying about it was absolutely accurate. This video is 100% accurate.
In order to help make easier for the viewer of the video to read and carefully consider each one of its main sources, each of which were passages within the published-online document “SARS-like WIV1-C0V poised for human emergence” scientific report on the research that ultimately produced covid-19, I present the links here, to both the HTML and the pdf versions of that scientific-research report, and then right below that, the cited passages:
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https://www.pnas.org/doi/10.1073/pnas.1517719113
https://www.pnas.org/doi/pdf/10.1073/pnas.1517719113
“SARS-like WIV1-C0V poised for human emergence”
14 March 2016
Focusing on SARS-like virus sequences isolated from Chinese horseshoe bats, the results indicate a significant threat posed by WIV1-CoV. [That “WI” refers to the Wuhan Institute of Virology, and the virus “WIV1-CoV” is “the prototype of SARS-CoV” the virus that causes covid-19 disease. “COVID-19 (novel coronavirus disease-2019) is the disease, SARS-CoV-2 is the virus.” So, these experiments were crucial to the development of the virus that causes covid-19.] Both full-length and chimeric WIV1-CoV readily replicated efficiently in human airway cultures and in vivo [in living organisms], suggesting capability of direct transmission to humans. In addition, while monoclonal antibody treatments prove effective, the SARS-based vaccine approach failed to confer protection. Together, the study indicates an ongoing threat posed by WIV1-related viruses and the need for continued study and surveillance [“surveillance” being a let’s-pretend term in such biowarfare-research-reports, pretending to be concerned to prevent instead of to create new diseases]. …
Although previously associated with upper respiratory infections, the emergence of severe acute respiratory coronavirus (SARS-CoV) in 2002–2003, and more recently, Middle East respiratory syndrome (MERS)-CoV underscores the threat of cross-species transmission leading to virulent pandemic viral infections (1, 2). …
The results indicate that viruses using WIV1-CoV [Wuhan Institute of Virology] spike are poised to emerge in human populations due to efficient replication in primary human airway epithelial cell cultures. However, additional adaptation, potentially independent of the spike protein receptor-binding domain, is required for pathogenesis and epidemic disease. Importantly, monoclonal antibody strategies against SARS were effective against WIV1-CoV spike unlike available vaccine approaches. Together, the results highlight the utility of developing platforms to evaluate circulating zoonotic viruses as threats for future emergence and epidemic potential. …
Using the SARS-CoV infectious clone as a template (7), we designed and synthesized a full-length infectious clone of WIV1-CoV consisting of six plasmids that could be enzymatically cut, ligated together, and electroporated into cells to rescue replication competent progeny virions (Fig. S1A). In addition to the full-length clone, we also produced WIV1-CoV chimeric virus that replaced the SARS spike with the WIV1 spike within the mouse-adapted backbone (WIV1-MA15, Fig. S1B). WIV1-MA15 incorporates the original binding and entry capabilities of WIV1-CoV, but maintains the backbone changes to mouse-adapted SARS-CoV. Importantly, WIV1-MA15 does not incorporate the Y436H mutation in spike that is required for SARS-MA15 pathogenesis (8). …
Replication in Primary Human Epithelial Cells. Next, we wanted to determine WIV-CoV replication potential in models of the human lung. Previous examination of WIV1-CoV recovered from bat samples demonstrated poor replication in A549 cells (5); however, replication of epidemic SARS-CoV is also poor in this cell type, potentially due to ACE2 expression levels (9).
Therefore, well-differentiated primary human airway epithelial cell (HAE) air–liquid interface cultures were infected with WIV1-MA15, WIV1-CoV, SARS-CoV Urbani, or SARS-CoV MA15. At 24 and 48 h postinfection, both WIV1-MA15 and WIV1-CoV produce robust infection in HAE cultures equivalent to the epidemic strain and mouse-adapted strains (Fig. 1D). Together, the data demonstrate that the WIV1-CoV spike can mediate infection of human airway cultures with no significant adaptation required.
